The electrocardiogram (ECG) records the electrical activity of the heart on the body surface, translating the heart's electrical signals into a standardized waveform of P waves, QRS complexes, and T waves. Deviations from the normal ECG pattern reveal arrhythmias, conduction blocks, myocardial infarction, and electrolyte abnormalities.
You already understand that cardiac muscle cells generate action potentials and that these electrical signals propagate through the heart in a coordinated sequence starting from the SA node. The electrocardiogram (ECG or EKG) is the clinical tool that lets us observe this electrical activity from outside the body, using electrodes placed on the skin. It does not measure the action potential of any single cell — instead, it detects the sum of all electrical activity across millions of cardiac cells at each instant, projected onto different recording axes called leads. The standard 12-lead ECG provides twelve different "viewing angles" of the same electrical events, giving a comprehensive picture of how depolarization and repolarization sweep through the heart.
The normal ECG tracing has three main deflections that map directly onto the conduction sequence you already know. The P wave represents atrial depolarization — the electrical wave spreading from the SA node across both atria. The QRS complex represents ventricular depolarization — the rapid, powerful wave traveling through the bundle of His, bundle branches, and Purkinje fibers to activate the thick ventricular muscle. The QRS is much larger than the P wave because the ventricular muscle mass is far greater. The T wave represents ventricular repolarization — the recovery phase as ventricular cells return to their resting membrane potential. Atrial repolarization also occurs, but it is hidden within the larger QRS complex and is not normally visible.
The intervals between these waves carry critical diagnostic information. The PR interval (from the start of the P wave to the start of the QRS) reflects conduction time through the atria and AV node — normally 120–200 ms. A prolonged PR interval indicates a conduction delay at the AV node. The QRS duration (normally < 120 ms) reflects how quickly the ventricles depolarize; a widened QRS suggests a bundle branch block or abnormal conduction pathway. The QT interval (from QRS onset to the end of the T wave) represents the total time for ventricular depolarization and repolarization; prolongation increases the risk of dangerous arrhythmias.
The power of the ECG lies in pattern recognition. A missing P wave before a QRS complex suggests the impulse did not originate from the SA node — possibly an ectopic ventricular beat or atrial fibrillation. An irregularly irregular rhythm with no identifiable P waves is the hallmark of atrial fibrillation, where the atria quiver chaotically instead of contracting in an organized manner. ST segment elevation (the flat segment between the QRS and T wave being pushed upward) is a classic sign of acute myocardial infarction — injured ventricular muscle creates abnormal current flow that distorts this baseline. Each deviation from the normal pattern tells a specific physiological story, making the ECG one of the most information-dense diagnostic tools in medicine.
No topics depend on this one yet.